Activation of Histone Deacetylase-6 (HDAC6) Induces Contractile Dysfunction through Derailment of -Tubulin Proteostasis in Experimental and Human Atrial Fibrillation

نویسندگان

  • Deli Zhang
  • MScChia-Tung Wu
  • XiaoYan Qi
  • Roelien A.M. Meijering
  • Femke Hoogstra-Berends
  • Artavazd Tadevosyan
  • Gunseli Cubukcuoglu Deniz
  • Serkan Durdu
  • Achmet Ruchan Akar
  • Ody C.M. Sibon
  • Stanley Nattel
  • Robert H. Henning
چکیده

Dept of Clinical Pharmacology, University Medical Center Groningen, University of Groningen, The Netherlands; Research Center and Dept of Medicine; Montreal Heart Institute and Université de Montréal, Montreal, Canada; Chang-Gung Memorial Hospital and ChangGung University, Taoyuan, Taiwan, Republic of China; Nyken BV, Groningen, The Netherlands; Ankara University Biotechnology Institute, Ankara, Turkey; Ankara University Stem Cell Institute, Ankara, Turkey; Dept of Cardiovascular Surgery, Ankara University School of Medicine, Turkey; Dept of Cell Biology, University Medical Center Groningen, University of Groningen, The Netherlands; Dept of Pharmacology, McGill University, Montreal, Canada

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Activation of histone deacetylase-6 induces contractile dysfunction through derailment of α-tubulin proteostasis in experimental and human atrial fibrillation.

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تاریخ انتشار 2013